At the same time, the secretion of reactive dentin by odontoblasts is accelerated, eventually leading to the formation of pulp stones or diffuse calcification and blocking the root canal system. In the defensive process, the formation of the calcification focus is centred on the degeneration and necrosis pulp cells. In the situation of tooth caries, periodontal disease, trauma and abrasion, and dental pulp produce a defensive reaction. Some scholars have proposed a relationship between periodontal disease and pulp calcification, and pulp calcification is a common manifestation in the healing process after a traumatic injury. Dental-pulp calcification can be considered a kind of active repair. Therefore, age-related calcifications are mostly located in the medullary cavity, canal orifice, and upper canal segment.
With age, secondary dentin accumulates gradually, pulp cavity and root canal become smaller, fat may deposit in the pulp, and calcification usually occurs at these deposits.
Between the ages of 20 and 70, these cells have been reported to decrease by 50%. believed that pulp calcification is an age-related disease. The aging process of tooth pulp leads to a decrease in fibroblast, odontoblast, and mesenchymal cells. Systemic factors include end-stage renal diseases, cardiovascular disease, and some long-term medications. Excessive forces caused by trauma and clenching, the presence of restorations, cavity preparation, and caries are common local factors. Local and systemic factors may contribute to the formation of dental pulp calcification. Glide path is the key to infection control in RCT, but in clinical practice, root canals are often blocked due to pulp calcification, which makes accessing and cleaning the root canal system more difficult. The Creative Commons Public Domain Dedication waiver ( ) applies to the data made available in this article, unless otherwise stated in a credit line to the data. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made.
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